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Age-related maculopathy (ARM) is a heterogeneous multifactorial disease and visual disability that presents as the most important cause of legal blindness in the elderly population. Since its definition and in the absence of a single effective treatment, it has been the subject of extensive research with the goal of identifying genetic determinants of susceptibility and of progression of ARM. Recently, these aims have largely been met by genome-wide association studies in large populations of diverse ancestry. A genome-wide association study was performed in 484 subjects of European descent from the Australian ‘Genetic Epidemiology of Age-related Maculopathy Study’; using an extended genome-wide SNP array, 80 single nucleotide polymorphisms associated with ARM were identified. In addition, genome-wide association studies with the most promising SNP and haplotype tagging SNPs were performed in two further Australian populations (332
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